Hyperglycemia-induced TET3 insufficiency is responsible for maternal transmission of glucose intolerance†

Wei Yan; Marisa S. Bartolomei

doi:10.1093/biolre/ioac115

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Wei Yan,^1,2,* Marisa S. Bartolomei^3,*

¹The Lundquist Institute for Biomedical Innovation at Harbor-UCLA Medical Center, Torrance, CA, USA
²Department of Medicine, David Geffen School of Medicine at UCLA, University of California, Los Angeles, CA, USA
³Department of Cell and Developmental Biology, Perelman School of Medicine, Epigenetics Institute, University of Pennsylvania, Philadelphia, PA, USA

^*Correspondence: Wei Yan, The Lundquist Institute at Harbor-UCLA, 1124 West Carson Street, Torrance, CA 90502. Tel: 775-338-985; E-mail: wei.yan@lundquist.org; Marisa S. Bartolomei, Department of Cell and Developmental Biology, University of Pennsylvania Perelman School of Medicine, 3400 Civic Center Blvd., Philadelphia, PA 19104. Tel: 215-898-9063; bartolom@pennmedicine.upenn.edu

No abstract for this paper is available.

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Wei Yan and Marisa S. Bartolomei "Hyperglycemia-induced TET3 insufficiency is responsible for maternal transmission of glucose intolerance," Biology of Reproduction 107(3), 676-678, (27 May 2022). https://doi.org/10.1093/biolre/ioac115

Received: 18 May 2022; Accepted: 22 May 2022; Published: 27 May 2022

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