BioOne.org will be down briefly for maintenance on 17 December 2024 between 18:00-22:00 Pacific Time US. We apologize for any inconvenience.
Open Access
How to translate text using browser tools
1 January 1982 COLOSTRUM DEFICIENCY IN MULE DEER FAWNS: IDENTIFICATION, TREATMENT AND INFLUENCE ON NEONATAL MORTALITY
DANIEL E. PARKINSON, ROBERT P. ELLIS, LON D. LEWIS
Author Affiliations +
Abstract

Glutaraldehyde coagulation test, zinc sulfate turbidity test, and total protein refractometry were adapted for use in detecting failure of passive transfer of colostral immunoglobulins to mule deer fawns (Odocoileus hemionus). The results of all three tests were similar. Serum total protein concentration was directly correlated to gamma globulin concentration and gave the best indication of morbidity and mortality. Thirteen of 13 fawns with serum total protein concentrations of 5 g/dl or less at 1 to 7 days of age developed diarrhea and died before 17 days of age. Only 1 of 14 fawns having a serum protein concentration above this level became sick and died. Seven of 13 fawns that had serum total protein concentrations of 5 g/dl or less, and that had already developed diarrhea, were given 20 ml of plasma per kg body weight. Although this increased their serum gamma globulin concentrations 0.3 g/dl, none survived. Administration of bovine colostrum to one fawn increased its serum gamma globulin concentration suggesting that mule deer fawns are able to absorb gamma globulins from bovine colostrum.

PARKINSON, ELLIS, and LEWIS: COLOSTRUM DEFICIENCY IN MULE DEER FAWNS: IDENTIFICATION, TREATMENT AND INFLUENCE ON NEONATAL MORTALITY1
DANIEL E. PARKINSON, ROBERT P. ELLIS, and LON D. LEWIS "COLOSTRUM DEFICIENCY IN MULE DEER FAWNS: IDENTIFICATION, TREATMENT AND INFLUENCE ON NEONATAL MORTALITY," Journal of Wildlife Diseases 18(1), 17-28, (1 January 1982). https://doi.org/10.7589/0090-3558-18.1.17
Received: 26 May 1981; Published: 1 January 1982
Back to Top